Muscle atrophy, ubiquitin-proteasome, and autophagic pathways in dysferlinopathy

Author:

Fanin Marina1,Nascimbeni Anna C.1,Angelini Corrado2

Affiliation:

1. Department of Neurosciences; University of Padova; Biomedical Campus “Pietro d'Abano,” via Giuseppe Orus 2B 35129 Padova Italy

2. Neuromuscular Department; IRCCS San Camillo Hospital; Lido-Venice Italy

Funder

Association Francaise Contre les Myopathies

Publisher

Wiley

Subject

Physiology (medical),Cellular and Molecular Neuroscience,Neurology (clinical),Physiology

Reference27 articles.

1. Identification of ubiquitin ligases required for skeletal muscle atrophy;Bodine;Science,2001

2. Autophagy: renovation of cells and tissues;Mizushima;Cell,2011

3. p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy;Pankiv;J Biol Chem,2007

4. Clinical utility of LC3 and p62 immunohistochemistry in diagnosis of drug-induced autophagic vacuolar myopathies: a case-control study;Lee;PLoS One,2012

5. FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells;Zhao;Cell Metab,2007

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号  京ICP备18003416号-3