High expression of the HMGB1-TLR4 axis and its downstream signaling factors in patients with Parkinson's disease and the relationship of pathological staging
Author:
Affiliation:
1. Department of Neonatologe; The Second Affiliated Hospital of Jiaxing University; Jiaxing China
Publisher
Wiley
Subject
Behavioral Neuroscience
Reference24 articles.
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2. HMGB1 translocation and release mediate cigarette smoke-induced pulmonary inflammation in mice through a TLR4/MyD88-dependent signaling pathway;Cheng;Molecular Biology of the Cell,2017
3. The danger-associated molecular pattern HMGB1 mediates the neuroinflammatory effects of methamphetamine;Frank;Brain Behavior & Immunity,2015
4. HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer's disease;Fujita;Scientific Reports,2016
5. HMGB1 acts on microglia Mac1 to mediate chronic neuroinflammation that drives progressive neurodegeneration;Gao;Journal of Neuroscience,2011
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