Inhibition of MDFI attenuates proliferation and glycolysis of Helicobacter pylori ‐infected gastric cancer cells by inhibiting Wnt/β‐catenin pathway
Author:
Affiliation:
1. Department of Gastroenterology The First Affiliated Hospital of Xi'an Jiaotong University Xi'an Shaanxi province China
Publisher
Wiley
Subject
Cell Biology,General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/cbin.11907
Reference24 articles.
1. Molecular Mechanism of Helicobacter pylori-Induced Gastric Cancer
2. Effect ofHelicobacter pylorion gastric epithelial cells
3. Review: Pathogenesis of Helicobacter pylori infection;Chmiela M.;Helicobacter,2019
4. CD36 inhibits β-catenin/c-myc-mediated glycolysis through ubiquitination of GPC4 to repress colorectal tumorigenesis
5. Helicobacter pylori CagA Protein Attenuates 5-Fu Sensitivity of Gastric Cancer Cells Through Upregulating Cellular Glucose Metabolism
Helicobacter pylori CagA Protein Attenuates 5-Fu Sensitivity of Gastric Cancer Cells Through Upregulating Cellular Glucose Metabolism
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1. The role of miR‐128 and MDFI in cardiac hypertrophy and heart failure: Mechanistic;Journal of Cellular and Molecular Medicine;2024-07
2. MDFI promotes the proliferation and tolerance to chemotherapy of colorectal cancer cells by binding ITGB4/LAMB3 to activate the AKT signaling pathway;Cancer Biology & Therapy;2024-02-20
3. Interactions between Helicobacter pylori infection and host metabolic homeostasis: A comprehensive review;Helicobacter;2023-10-23
4. Butyrate prevents the migration and invasion, and aerobic glycolysis in gastric cancer via inhibiting Wnt/β‐catenin/c‐Myc signaling;Drug Development Research;2023-02-13
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