Subtle phenotypes of Mowat–Wilson syndrome in a patient with a novel ZEB2 C‐ZF domain variant

Author:

Kuroda Yukiko1ORCID,Naruto Takuya2,Kurosawa Kenji1ORCID

Affiliation:

1. Division of Medical Genetics Kanagawa Children's Medical Center Yokohama Kanagawa Japan

2. Clinical Research Institute Kanagawa Children's Medical Center Yokohama Kanagawa Japan

Funder

Japan Agency for Medical Research and Development

Publisher

Wiley

Reference11 articles.

1. Letter regarding the article "Extending the phenotype of recurrent rearrangements of 16p11.2: Deletions in mentally retarded patients without autism and in normal individuals (Bijlsma et al., 2009)" and the diagnosis of coexisting Mowat–Wilson syndrome in a patient with 16p11.2 deletion;Amor D. J.;European Journal of Medical Genetics,2018

2. De novo variants in neurodevelopmental disorders‐experiences from a tertiary care center;Brunet, T., Jech, R., Brugger, M., Kovacs, R., Alhaddad, B., Leszinski, G., Riedhammer, K. M., Westphal, D. S., Mahle, I., Mayerhanser, K., Skorvanek, M., Weber, S., Graf, E., Berutti, R., Necpál, J., Havránková, P., Pavelekova, P., Hempel, M., Kotzaeridou, U., … Wagner, M.;Clinical Genetics,2021

3. ZFHX1B mutations in patients with Mowat‐Wilson syndrome;Dastot‐Le Moal, F., Wilson, M., Mowat, D., Collot, N., Niel, F., & Goossens, M.;Human Mutations,2007

4. ZEB2 zinc‐finger missense mutations lead to hypomorphic alleles and a mild Mowat–Wilson syndrome;Ghoumid J.;Human Molecular Genetics,2013

5. Phenotype and genotype of 87 patients with Mowat–Wilson syndrome and recommendations for care;Ivanovski I.;Genetics in Medicine,2018

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