Protein surveillance machinery in brains with spinocerebellar ataxia type 3: Redistribution and differential recruitment of 26S proteasome subunits and chaperones to neuronal intranuclear inclusions-Reference-Cited by-同舟云学术

Protein surveillance machinery in brains with spinocerebellar ataxia type 3: Redistribution and differential recruitment of 26S proteasome subunits and chaperones to neuronal intranuclear inclusions

Published:2002-02-27 Issue:3 Volume:51 Page:302-310
ISSN:0364-5134
Container-title:Annals of Neurology
language:en
Short-container-title:Ann Neurol.

Author:

Schmidt Thorsten,Lindenberg Katrin S.,Krebs Antje,Schöls Ludger,Laccone Franco,Herms Jochen,Rechsteiner Martin,Riess Olaf,Landwehrmeyer G. Bernhard

Funder

Deutsche Forschungsgemeinschaft

German Hereditary Ataxia Foundation (to OR)

Graduierten-Kolleg für Molekulare Medizin (to KSL)

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Reference50 articles.

1. 1;Kawaguchi;Nat Genet,1994

2. Spinocerebellar ataxia 3 and Machado-Joseph disease: clinical, molecular, and neuropathological features;Dürr;Ann Neurol,1996

3. Relations between genotype and phenotype in German patients with the Machado-Joseph disease mutation;Schöls;J Neurol Neurosurg Psychiatry,1996

4. Machado-Joseph disease mutations as the genetic basis of most spinocerebellar ataxias in Germany;Schöls;J Neurol Neurosurg Psychiatry,1995

5. Glutamine repeats and neurodegeneration;Zoghbi;Annu Rev Neurosci,2000

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