The putative involvement of actin-binding proteins and cytoskeleton proteins in pathological mechanisms of ketamine cystitis-Revealed by a prospective pilot study using proteomic approaches
Author:
Affiliation:
1. Department of Medical Research; Buddhist Tzu Chi General Hospital; Hualien 970 Taiwan
2. Department of Urology; Buddhist Tzu Chi General Hospital, Tzu Chi University; Hualien 970 Taiwan
Funder
National Science Council
Publisher
Wiley
Subject
Clinical Biochemistry
Reference39 articles.
1. Ketamine-associated ulcerative cystitis: a new clinical entity;Shahani;Urology,2007
2. The destruction of the lower urinary tract by ketamine abuse: a new syndrome;Chu;BJU Int.,2008
3. Increased apoptosis and suburothelial inflammation in patients with ketamine-related cystitis: a comparison with non-ulcerative interstitial cystitis and controls;Lee;BJU Int.,2013
4. Involvement of purinergic neurotransmission in ketamine induced bladder dysfunction;Meng;J. Urol.,2011
5. Nerve hyperplasia: a unique feature of ketamine cystitis;Baker;Acta Neuropathol. Commun.,2013
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1. Pathophysiology, clinical presentation, and management of ketamine-induced cystitis;Tzu Chi Medical Journal;2023
2. Association of urinary ketamine and APOA1 levels with bladder dysfunction in ketamine abusers revealed via proteomics and targeted metabolite analyses;Scientific Reports;2021-05-05
3. Smaller bladder capacity and stronger bladder contractility in patients with ketamine cystitis are associated with elevated TRPV1 and TRPV4;Scientific Reports;2021-03-04
4. Regulation of cytochrome P450 gene expression by ketamine: a review;Expert Opinion on Drug Metabolism & Toxicology;2018-06-17
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