Autoantibodies targeting glomerular annexin A2 identify patients with proliferative lupus nephritis

Author:

Caster Dawn J.12,Korte Erik A.3,Merchant Michael L.1,Klein Jon B.12,Wilkey Daniel W.1,Rovin Brad H.4,Birmingham Dan J.4,Harley John B.56,Cobb Beth L.5,Namjou Bahram5,McLeish Kenneth R.12,Powell David W.13

Affiliation:

1. Department of Medicine, University of Louisville School of Medicine; Louisville KY USA

2. Robley Rex Veterans Affairs Medical Center; Louisville KY USA

3. Department of Biochemistry and Molecular Biology; University of Louisville School of Medicine; Louisville KY USA

4. Department of Medicine; the Ohio State University; Columbus OH USA

5. Center for Autoimmune Genomics and Etiology; Department of Pediatrics; Cincinnati Children's Hospital Medical Center and the University of Cincinnati; Cincinnati OH USA

6. US Department of Veterans Affairs Medical Center; Cincinnati OH USA

Funder

National Institutes of Health

Juvenile Diabetes Research Foundation international

Publisher

Wiley

Subject

Clinical Biochemistry

Reference63 articles.

1. Systemic lupus erythematosus;Tsokos;N. Engl. J. Med.,2011

2. Serology of Lupus erythematosus: correlation between immunopathological features and clinical aspects;Cozzani;Autoimmune Dis.,2014

3. Membranous (class V) renal disease in systemic lupus erythematosus may be more common than previously reported: results of a 6-year retrospective analysis;Bhinder;Am. J. Med. Sci.,2010

4. The classification of glomerulonephritis in systemic lupus erythematosus revisited;Weening;Kidney Int.,2004

5. Multiple autoantibodies form the glomerular immune deposits in patients with systemic lupus erythematosus;Mannik;J. Rheumatol.,2003

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