Epigenetic mechanism causes Wnt9b deficiency and nonsyndromic cleft lip and palate in the A/WySn mouse strain-Reference-Cited by-同舟云学术

Epigenetic mechanism causes Wnt9b deficiency and nonsyndromic cleft lip and palate in the A/WySn mouse strain

Published:2014-09-25 Issue:10 Volume:100 Page:772-788
ISSN:1542-0752
Container-title:Birth Defects Research Part A: Clinical and Molecular Teratology
language:en
Short-container-title:Birth Defects Research Part A: Clinical and Molecular Teratology

Author:

Juriloff Diana M.¹,Harris Muriel J.¹,Mager Dixie L.¹²,Gagnier Liane¹²

Affiliation:

1. Department of Medical Genetics; University of British Columbia; Vancouver British Columbia Canada

2. Terry Fox Laboratory; British Columbia Cancer Agency; Vancouver British Columbia Canada

Funder

Canadian Institutes of Health Research

Publisher

Wiley

Subject

Developmental Biology,Embryology,General Medicine,Pediatrics, Perinatology and Child Health

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/bdra.23320/fullpdf

Reference70 articles.

1. Epigenomic disruption: the effects of early developmental exposures;Bernal;Birth Defects Res A Clin Mol Teratol,2010

2. Wnt9b plays a central role in the regulation of mesenchymal to epithelial transitions underlying organogenesis of the mammalian urogenital system;Carroll;Dev Cell,2005

3. Variation in WNT genes is associated with non-syndromic cleft lip with or without cleft palate;Chiquet;Hum Mol Genet,2008

4. Functional analysis of the long terminal repeats of intracisternal A-particle genes: sequences within the U3 region determine both the efficiency and direction of promoter activity;Christy;Mol Cell Biol,1988

5. A comparative study of development during primary palate formation in A/WySn, C57BL/6, and their F1 crosses;Ciriani;J Craniofac Genet Dev Biol,1986

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