Anisomycin abrogates repression of protooncogenec-fos transcription in E1A + cHa-ras-transformed cells through activation of MEK/ERK kinase cascade
Author:
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference32 articles.
1. Selective repression of c-fos gene transcription in rat embryo fibroblasts transformed by oncogenes E1A and cHa-ras
2. G1/S Arrest Induced by Histone Deacetylase Inhibitor Sodium Butyrate in E1A + Ras-transformed Cells Is Mediated through Down-regulation of E2F Activity and Stabilization of β-Catenin
3. Immediate-early gene induction by the stresses anisomycin and arsenite in human osteosarcoma cells involves MAPK cascade signaling to Elk-1, CREB and SRF
4. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
5. The Dual Specificity Mitogen-activated Protein Kinase Phosphatase-1 and -2 Are Induced by the p42/p44MAPK Cascade
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2. The effects of the HDAC inhibitor sodium butyrate on the expression of repair genes Rad51 and XRCC5 in fibroblast lines mEras-Waf1+/+ and mEras-Waf1–/–;Cell and Tissue Biology;2017-03
3. MKP-1 protein phosphatase participates in c-fos gene derepression in E1A and cHA-ras oncogene transformed fibroblasts under stress conditions;Cell and Tissue Biology;2014-03
4. Low-dose anisomycin is sufficient to alter the bio-behaviors of Jurkat T cells;Open Life Sciences;2013-12-01
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