A20 overexpression exerts protective effects on podocyte injury in lupus nephritis by downregulating UCH‐L1
Author:
Affiliation:
1. Department of Nephrology Xuzhou Central Hospital, Medical College of Southeast University Xuzhou Jiangsu China
2. School of Management, Zhejiang University Hangzhou China
3. Institute of Nephrology, Zhongda Hospital, Southeast University China
Funder
National Natural Science Foundation of China
Natural Science Foundation of Jiangsu Province
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28282
Reference44 articles.
1. Hypomorphic A20 expression confers susceptibility to psoriasis
2. Treatment of Proliferative and Membranous Lupus Nephritis: Review of Key Clinical Trials
3. Podocyte foot process effacement is not correlated with the level of proteinuria in human glomerulopathies
4. US patients of Hispanic and African ancestry develop lupus nephritis early in the disease course: data from LUMINA, a multiethnic US cohort (LUMINA LXXIV)
5. UCH-L1 Expressed by Podocytes: a Potentially Therapeutic Target for Lupus Nephritis?
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