Targeting protein–protein interactions to rescue Δf508‐cftr: a novel corrector approach to treat cystic fibrosis
Author:
Affiliation:
1. Instituto de Biología Molecular y CelularUniversidad Miguel HernándezAlicanteSpain
Publisher
EMBO
Subject
Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/emmm.201303301
Reference10 articles.
1. Effect of VX-770 in Persons with Cystic Fibrosis and the G551D-CFTRMutation
2. CYSTIC FIBROSIS OF THE PANCREAS AND ITS RELATION TO CELIAC DISEASE
3. Results of a phase IIa study of VX-809, an investigational CFTR corrector compound, in subjects with cystic fibrosis homozygous for theF508del-CFTRmutation
4. Novel pharmacological strategies to treat cystic fibrosis
5. Cystic fibrosis: Exploiting its genetic basis in the hunt for new therapies
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3. Elucidating Protein-protein Interactions Through Computational Approaches and Designing Small Molecule Inhibitors Against them for Various Diseases;Current Topics in Medicinal Chemistry;2018-12-31
4. Rattlesnake Phospholipase A2 Increases CFTR-Chloride Channel Current and Corrects ∆ F508CFTR Dysfunction: Impact in Cystic Fibrosis;Journal of Molecular Biology;2016-07
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