Targeting aurora kinases limits tumour growth through DNA damage‐mediated senescence and blockade of NF‐κB impairs this drug‐induced senescence-Reference-Cited by-同舟云学术

Targeting aurora kinases limits tumour growth through DNA damage‐mediated senescence and blockade of NF‐κB impairs this drug‐induced senescence

Published:2012-11-25 Issue:1 Volume:5 Page:149-166
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Liu Yan¹²,Hawkins Oriana E.¹²,Su Yingjun¹²,Vilgelm Anna E.¹²,Sobolik Tammy¹²,Thu Yee‐Mon¹²,Kantrow Sara³,Splittgerber Ryan C.²,Short Sarah¹²,Amiri Katayoun I.²,Ecsedy Jeffery A.⁴,Sosman Jeffery A.⁵,Kelley Mark C.⁶,Richmond Ann¹²

Affiliation:

1. Department of Veterans Affairs, Tennessee Valley Healthcare System, Nashville, TN, USA

2. Department of Cancer Biology, Vanderbilt University Medical Center, Nashville, TN, USA

3. Division of Dermatology, Vanderbilt University Medical Center, Nashville, TN, USA

4. Millennium Pharmaceuticals, Inc., Cambridge, MA, USA

5. Division of Hematology/Oncology, Department of Medicine, Vanderbilt University Medical Center, Nashville, TN, USA

6. Division of Surgical Oncology, Department of Surgery, Vanderbilt University School of Medicine, Nashville, TN, USA

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/emmm.201201378

Reference56 articles.

1. Chemokine Signaling via the CXCR2 Receptor Reinforces Senescence

2. Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblasts

3. Ink4a/Arf and Oncogene-Induced Senescence Prevent Tumor Progression during Alternative Colorectal Tumorigenesis

4. A line of non-tumorigenic mouse melanocytes, syngeneic with the B16 melanoma and requiring a tumour promoter for growth

5. Oncogene-induced senescence as an initial barrier in lymphoma development

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