A CAG repeat polymorphism of KCNN3 predicts SK3 channel function and cognitive performance in schizophrenia-Reference-Cited by-同舟云学术

A CAG repeat polymorphism of KCNN3 predicts SK3 channel function and cognitive performance in schizophrenia

Published:2011-03-24 Issue:6 Volume:3 Page:309-319
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Grube Sabrina¹,Gerchen Martin F.¹,Adamcio Bartosz¹,Pardo Luis A.²,Martin Sabine²³,Malzahn Dörthe⁴,Papiol Sergi¹³⁵,Begemann Martin¹,Ribbe Katja¹,Friedrichs Heidi¹,Radyushkin Konstantin A¹³,Müller Michael³⁶,Benseler Fritz⁷,Riggert Joachim⁸,Falkai Peter³⁹,Bickeböller Heike⁴,Nave Klaus‐Armin³⁵,Brose Nils³⁷,Stühmer Walter²³,Ehrenreich Hannelore¹³

Affiliation:

1. Divison of Clinical Neuroscience, Max Planck Institute of Experimental Medicine, Göttingen, Germany

2. Department of Molecular Biology of Neuronal Signals, Max Planck Institute of Experimental Medicine, Göttingen, Germany

3. DFG Research Center for Molecular Physiology of the Brain (CMPB), Göttingen, Germany

4. Department of Genetic Epidemiology, Medical School, Georg‐August‐University, Göttingen, Germany

5. Department of Neurogenetics, Max Planck Institute of Experimental Medicine, Göttingen, Germany

6. Department of Neuro‐ and Sensory Physiology, Georg‐August‐University, Göttingen, Germany

7. Department of Molecular Neurobiology, Max Planck Institute of Experimental Medicine, Göttingen, Germany

8. Department of Transfusion Medicine, Georg‐August‐University, Göttingen, Germany

9. Department of Psychiatry and Psychotherapy, Georg‐August‐University, Göttingen, Germany

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/emmm.201100135

Reference43 articles.

1. Mapping of hKCa3 to chromosome 1q21 and investigation of linkage of CAG repeat polymorphism to schizophrenia

2. Modification of Cognitive Performance in Schizophrenia by Complexin 2 Gene Polymorphisms

3. Small-conductance, Ca2+-activated K+ channel SK3 generates age-related memory and LTP deficits

4. Respiration and Parturition Affected by Conditional Overexpression of the Ca 2+ -Activated K + Channel Subunit, SK3

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