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Endoplasmic reticulum stress activation during total knee arthroplasty

  • Published:2013-08 Issue:3 Volume:1 Page:
  • ISSN:2051-817X
  • Container-title:Physiological Reports
  • language:en
  • Short-container-title:Physiol Rep

Author:

Hocker Austin D.1,Boileau Ryan M.1,Lantz Brick A.2,Jewett Brian A.2,Gilbert Jeffrey S.1,Dreyer Hans C.1

Affiliation:

1. Department of Human Physiology; University of Oregon; Eugene Oregon

2. Slocum Center for Orthopedics and Sports Medicine; Eugene Oregon

Funder

National Center for Medical Rehabilitation and Research

National Institute of Child Health and Human Development (NICHD)

Office of the Director (OD)

NIH

Publisher

Wiley

Subject

Physiology (medical),Physiology

Reference57 articles.

1. Intracellular calcium concentration during hypoxia and metabolic inhibition in mammalian ventricular muscle;Allen;J. Physiol.,1983

2. Skeletal muscle damage during tourniquet-induced ischaemia. The initial step towards atrophy after orthopaedic surgery?;Appell;Eur. J. Appl. Physiol Occup. Physiol.,1993

3. TRB3 function in cardiac endoplasmic reticulum stress;Avery;Circ. Res.,2010

4. MAFbx, MuRF1, and the stress-activated protein kinases are upregulated in muscle cells during total knee arthroplasty;Bailey;Am. J. Physiol. Regul. Integr. Comp. Physiol.,2012

5. The relationship of strength to function in the older adult;Brown;J. Gerontol. A Biol. Sci. Med. Sci.,1995
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