Nuclear translocation of nuclear factor kappa B is regulated by G protein signaling pathway in arsenite-induced apoptosis in HBE cell line

Author:

Chen Chengzhi12,Gu Shiyan1,Jiang Xuejun12,Zhang Zunzhen1

Affiliation:

1. Department of Occupational and Environmental Health; West China School of Public Health, Sichuan University; Chengdu Sichuan People's Republic of China

2. Department of Occupational and Environmental Health; School of Public Health and Management, Chongqing Medical University; Chongqing People's Republic of China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Reference55 articles.

1. Low dose and long term toxicity of sodium arsenite caused caspase dependent apoptosis based on morphology and biochemical character;Abnosi;Cell J,2012

2. The paradox of arsenic: Molecular mechanisms of cell transformation and chemotherapeutic effects;Bode;Crit Rev Oncol Hematol,2002

3. Apoptosis: Overview and signal transduction pathways;Bredesen;J Neurotrauma,2000

4. The role of apoptosis in cancer development and treatment response;Brown;Nat Rev Cancer,2005

5. Signaling from toxic metals to NF-kappaB and beyond: Not just a matter of reactive oxygen species;Chen;Environ Health Perspect,2002

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