Activation ofPrn-p gene and stable transfection ofPrn-p cDNA in leukemia MEL and neuroblastoma N2a cells increased production of PrPC but not prevented DNA fragmentation initiated by serum deprivation-Reference-Cited by-同舟云学术

Activation ofPrn-p gene and stable transfection ofPrn-p cDNA in leukemia MEL and neuroblastoma N2a cells increased production of PrPC but not prevented DNA fragmentation initiated by serum deprivation

Published:2007 Issue:2 Volume:211 Page:551-559
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J. Cell. Physiol.

Author:

Gougoumas Dimitrios D.,Vizirianakis Ioannis S.,Triviai Ioanna N.,Tsiftsoglou Asterios S.

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference37 articles.

1. Pharmacological promotion of inclusion formation: A therapeutic approach for Huntington's and Parkinson's diseases

2. Prion Protein Protects Human Neurons against Bax-mediated Apoptosis

3. Interaction of PrP with NRAGE, a protein involved in neuronal apoptosis

4. The cellular prion protein binds copper in vivo

5. Prion diseases — close to effective therapy?

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