The balance between cathepsin C and cystatin F controls remyelination in the brain ofPlp1-overexpressing mouse, a chronic demyelinating disease model

Author:

Shimizu Takahiro1,Wisessmith Wilaiwan12,Li Jiayi13,Abe Manabu4,Sakimura Kenji4,Chetsawang Banthit2,Sahara Yoshinori5,Tohyama Koujiro56,Tanaka Kenji F.17,Ikenaka Kazuhiro13ORCID

Affiliation:

1. Division of Neurobiology and Bioinformatics; National Institute for Physiological Sciences; Okazaki Japan

2. Research Center for Neuroscience, Institute of Molecular Biosciences, Mahidol University; Salaya Nakhonpathom Thailand

3. Department of Physiological Sciences; Graduate University for Advanced Studies (SOKENDAI); Okazaki Japan

4. Brain Research Institute, Niigata University; Niigata Japan

5. Department of Physiology; Iwate Medical University School of Dentistry; Iwate Japan

6. Center for Electron Microscopy and Bio-Imaging Research, Iwate Medical University; Iwate Japan

7. Department of Neuropsychiatry; Keio University; Tokyo Japan

Funder

Grants-in-Aid for Scientific Research on Innovative Areas: “Foundation of Synapse and Neurocircuit Pathology

Glial Assembly: a new regulatory machinery of brain function and disorders

Thailand Research Fund

Akio Suzumura and Tetsuya Mizuno in the Department of Neuroimmunology, Research Institute of Environmental Medicine, Nagoya University, Japan

Yasuo Uchiyama and Masato Koike in the School of Medicine, Juntendo University, Japan

Masahiro Shibata in Niigata University, Japan

Maya Yamazaki (current affiliation: Niccole lab at the University of California San Francisco, USA)

Japan Society for the Promotion of Science

Grant-in-Aid for Strategic Medical Science Research for private university

Ministry of Education, Culture, Sports, Science and Technology

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

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