TRPV4 channels contribute to calcium transients in astrocytes and neurons during peri-infarct depolarizations in a stroke model-Reference-Cited by-同舟云学术

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TRPV4 channels contribute to calcium transients in astrocytes and neurons during peri-infarct depolarizations in a stroke model

Published:2017-06-22 Issue:9 Volume:65 Page:1550-1561
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Rakers Cordula¹,Schmid Matthias¹²,Petzold Gabor C.¹³^ORCID

Affiliation:

1. German Center for Neurodegenerative Diseases (DZNE); Sigmund-Freud-Street 27 Bonn 53127 Germany

2. Department of Medical Biometry; Informatics and Epidemiology, University Hospital Bonn; Sigmund-Freud-Street 25 Bonn 53127 Germany

3. Department of Neurology; University Hospital Bonn; Sigmund-Freud-Street 25 Bonn 53127 Germany

Funder

Else Kröner-Fresenius-Stiftung

Deutsche Forschungsgemeinschaft

European Union (EU)

Joint Programme - Neurodegenerative Disease Research (JPND)

EU Horizon 2020 Research and Innovation Program

DZNE

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/glia.23183/fullpdf

Reference41 articles.

1. A solution to dependency: Using multilevel analysis to accommodate nested data;Aarts;Nature Neuroscience,2014

2. Aquaporins in cerebrovascular disease: A target for treatment of brain edema?;Badaut;Cerebrovascular Diseases,2011

3. Ca2+ signals and neuronal death in brain ischemia;Bano;Stroke,2007

4. Expression and functional characterization of transient receptor potential vanilloid-related channel 4 (TRPV4) in rat cortical astrocytes;Benfenati;Neuroscience,2007

5. An aquaporin-4/transient receptor potential vanilloid 4 (AQP4/TRPV4) complex is essential for cell-volume control in astrocytes;Benfenati;Proceedings of the National Academy of Sciences of the United States of America,2011

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