Neobavaisoflavone protects osteoblasts from dexamethasone‐induced oxidative stress by upregulating the CRNDE ‐mediated Nrf2/ HO ‐1 signaling pathway-Reference-Cited by-同舟云学术

Neobavaisoflavone protects osteoblasts from dexamethasone‐induced oxidative stress by upregulating the CRNDE ‐mediated Nrf2/ HO ‐1 signaling pathway

Published:2021-03-13 Issue:7 Volume:82 Page:1044-1054
ISSN:0272-4391
Container-title:Drug Development Research
language:en
Short-container-title:Drug Dev Res

Author:

Zhu Zhonglian¹^ORCID,Wang Xuyi¹,Wang Zhaodong¹,Zhao Zhi¹,Zhou Pinghui¹,Gao Xubin¹

Affiliation:

1. Department of Orthopedics The First Affiliated Hospital of Bengbu Medical College; Anhui Key Laboratory of Tissue Transplantation, Bengbu Medical College Bengbu China

Publisher

Wiley

Subject

Drug Discovery

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ddr.21811

Reference41 articles.

1. A Review of Osteoporosis in the Older Adult

2. Post-ischemic brain damage: pathophysiology and role of inflammatory mediators

3. Neobavaisoflavone inhibits osteoclastogenesis through blocking RANKL signalling‐mediated TRAF6 and c‐Src recruitment and NF‐κB, MAPK and Akt pathways

4. Thymoma‐associated myasthenia gravis : Clinical features and predictive value of antiacetylcholine receptor antibodies in the risk of recurrence of thymoma

5. Dexamethasone induces osteoblast apoptosis through ROS-PI3K/AKT/GSK3β signaling pathway

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