Lesion-specific pattern of immunocytochemical distribution of growth-associated protein B-50 (GAP-43) in the cerebellum of weaver and PCD-mutant mice: Lack of B-50 involvement in neuroplasticity of Purkinje cell terminals?
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
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1. Long-Term Injured Purkinje Cells Are Competent for Terminal Arbor Growth, but Remain Unable to Sustain Stem Axon Regeneration;Experimental Neurology;2002-07
2. Expression of synaptic proteins in the developing rat cerebellum following ionizing radiation;International Journal of Developmental Neuroscience;1999-07
3. B-50, the growth associated protein-43: modulation of cell morphology and communication in the nervous system;Progress in Neurobiology;1997-12
4. Diverse effects of Purkinje cell loss on deep cerebellar and vestibular nuclei neurons in Purkinje cell degeneration mutant mice: A possible compensatory mechanism;The Journal of Comparative Neurology;1997-08-11
5. Differential number of glycine- and GABA-immunopositive neurons and terminals in the deep cerebellar nuclei of normal and Purkinje cell degeneration mutant mice;The Journal of Comparative Neurology;1997-06-16
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