Sustained Release of TPCA‐1 from Silk Fibroin Hydrogels Preserves Keratocyte Phenotype and Promotes Corneal Regeneration by Inhibiting Interleukin‐1 β Signaling-Reference-Cited by-全球学者库

Sustained Release of TPCA‐1 from Silk Fibroin Hydrogels Preserves Keratocyte Phenotype and Promotes Corneal Regeneration by Inhibiting Interleukin‐1 β Signaling

Published:2020-08-02 Issue:17 Volume:9 Page:2000591
ISSN:2192-2640
Container-title:Advanced Healthcare Materials
language:en
Short-container-title:Adv. Healthcare Mater.

Author:

Zhang Wei¹²³^ORCID,Chen Jialin¹²³,Qu Mingli⁴,Backman Ludvig J.²,Zhang Aini¹,Liu Haoyang¹,Zhang Xiaoping⁵,Zhou Qingjun⁴,Danielson Patrik²⁶^ORCID

Affiliation:

1. School of Medicine Southeast University Nanjing 210009 China

2. Department of Integrative Medical Biology, Anatomy Umeå University Umeå SE‐901 87 Sweden

3. Jiangsu Key Laboratory for Biomaterials and Devices Southeast University Nanjing 210096 China

4. State Key Laboratory Cultivation Base Shandong Provincial Key Laboratory of Ophthalmology, Shandong Eye Institute Shandong First Medical University & Shandong Academy of Medical Sciences Qingdao 266071 China

5. Ophthalmology Department the Affiliated Hospital of Qingdao University Qingdao 266003 China

6. Department of Clinical Sciences, Ophthalmology Umeå University Umeå SE‐901 87 Sweden

Funder

National Natural Science Foundation of China

Natural Science Foundation of Jiangsu Province

Fundamental Research Funds for the Central Universities

Svenska Läkaresällskapet

Korea Military Academy

Publisher

Wiley

Subject

Pharmaceutical Science,Biomedical Engineering,Biomaterials

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/adhm.202000591

Reference36 articles.

1. Progress in Corneal Stromal Repair: From Tissue Grafts and Biomaterials to Modular Supramolecular Tissue-Like Assemblies

2. World Health Organization Priority eye diseases https://www.who.int/blindness/causes/priority/en/index8.html(accessed February2020).

3. Progress in corneal wound healing

4. Understanding the mechanism of IL-1β secretion

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