Secretion of granulocyte-macrophage colony-stimulating factor by human blood monocytes is stimulated by engagement of Fcγ receptors type I by solid-phase immunoglobulins requiring high-affinity Fc-Fcγ receptor type I interactions-Reference-Cited by-同舟云学术

Secretion of granulocyte-macrophage colony-stimulating factor by human blood monocytes is stimulated by engagement of Fcγ receptors type I by solid-phase immunoglobulins requiring high-affinity Fc-Fcγ receptor type I interactions

Published:1992-07 Issue:7 Volume:22 Page:1681-1685
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Herrmann Friedhelm,de Vos Sven,Brach Marion,Riedel Detlef,Lindemann Albrecht,Mertelsmann Roland

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference31 articles.

1. The Human Hematopoietic Colony-Stimulating Factors

2. Tumor necrosis factor (TNF)-alpha but not TNF-beta induces secretion of colony stimulating factor for macrophages (CSF-1) by human monocytes

3. Expression of the macrophage-specific colony-stimulating factor in human monocytes treated with granulocyte-macrophage colony-stimulating factor

4. Interleukin-1 (22-K factor) induces release of granulocyte-macrophage colony-stimulating activity from human mononuclear phagocytes

Cited by 6 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Soluble Immune Complexes Shift the TLR-Induced Cytokine Production of Distinct Polarized Human Macrophage Subsets towards IL-10;PLoS ONE;2012-04-26

2. Activation of FcγRI on Monocytes Triggers Differentiation into Immature Dendritic Cells That Induce Autoreactive T Cell Responses;The Journal of Immunology;2009-07-27

3. Different types of FC γ -receptors are involved in anti-Lewis Y antibody induced effector functions in vitro;British Journal of Cancer;2000-01

4. Enhanced Production of an Egf-Like Growth Factor by Parenchymal Macrophages Following Bleomycin-Induced Pulmonary Injury;Experimental Lung Research;1997-01

5. The Expression and Regulation of G-CSF and GM-CSF;Acute Leukemias V;1996