Lack of detectable defect in DNA double-strand break repair and DNA-dependent protein kinase activity in radiosensitive human severe combined immunodeficiency fibroblasts-Reference-Cited by-同舟云学术

Lack of detectable defect in DNA double-strand break repair and DNA-dependent protein kinase activity in radiosensitive human severe combined immunodeficiency fibroblasts

Published:1996-05 Issue:5 Volume:26 Page:1118-1122
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Nicolas Nathalie,Finnie Nicholas J.,Cavazzana-Calvo Marina,Papadopoulo Dora,Le Deist Françoise,Fischer Alain,Jackson Stephen P.,de Villartay Jean-Pierre

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference28 articles.

1. VDJ recombination

2. Cleavage at a V(D)J recombination signal requires only RAG1 and RAG2 proteins and occurs in two steps

3. RAG-1-deficient mice have no mature B and T lymphocytes

4. RAG-2-deficient mice lack mature lymphocytes owing to inability to initiate V(D)J rearrangement

5. The SCID Mouse Mutant: Definition, Characterization, and Potential Uses

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2. Severe Combined Immune Deficiency with Absence of B and T Lymphocytes (T − B − NK + SCIDs): The Key Function of V(D)J Recombination for Lymphocyte Development;Encyclopedia of Immunobiology;2016

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