Genetic requirements for acceleration of diabetes in non-obese diabetic mice expressing interleukin-2 in islet β-cells-Reference-Cited by-同舟云学术

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Genetic requirements for acceleration of diabetes in non-obese diabetic mice expressing interleukin-2 in islet β-cells

Published:1994-10 Issue:10 Volume:24 Page:2535-2541
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Allison Janette,McClive Peter,Oxbrow Leonie,Baxter Alan,Morahan Grant,Miller Jacques F. A. P.

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference52 articles.

1. An Overview on Possible Mechanisms of Destruction of the Insulin-Producing Beta Cell

2. The involvement of Ly 2+ T cells in beta cell destruction

3. Major histocompatibility complex class I molecules are required for the development of insulitis in non-obese diabetic mice

4. β2-Microglobulin–Deficient NOD Mice Do Not Develop Insulitis or Diabetes

5. Major Histocompatibility Complex Class I-Deficient NOD-B2mnull Mice are Diabetes and Insulitis Resistant

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4. Loss of parity between IL-2 and IL-21 in the NOD Idd3 locus;Proceedings of the National Academy of Sciences;2009-10-30

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