A cross-species functional interaction between the murine major histocompatibility complex class I α3 domain and human CD8 revealed by peptide-specific cytotoxic T lymphocytes-Reference-Cited by-同舟云学术

A cross-species functional interaction between the murine major histocompatibility complex class I α3 domain and human CD8 revealed by peptide-specific cytotoxic T lymphocytes

Published:1992-06 Issue:6 Volume:22 Page:1643-1646
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Moots Robert J.,Samberg Nancy L.,Pazmany Laszlo,Frelinger Jeffrey A.,McMichael Andrew J.,Stauss Hans J.

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference26 articles.

1. Cytotoxic T lymphocytes recognize a fragment of influenza virus matrix protein in association with HLA-A2

2. The epitopes of influenza nucleoprotein recognized by cytotoxic T lymphocytes can be defined with short synthetic peptides

3. Adhesion receptors of the immune system

4. Human T cell antigens involved in cytotoxicity against allogeneic or autologous chemically modified targets. Association of the Leu 2a/T8 antigen with effector-target cell binding and of the T3/Leu 4 antigen with triggering

5. A binding site for the T-cell co-receptor CD8 on the α3 domain of HLA-A2

Cited by 12 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. MAIT cell-MR1 reactivity is highly conserved across multiple divergent species;Journal of Biological Chemistry;2024-06

2. MHC class II‐deficient mice allow functional human CD4⁺ T‐cell development;European Journal of Immunology;2023-05-12

3. GVHD Pathogenesis, Prevention and Treatment: Lessons From Humanized Mouse Transplant Models;Frontiers in Immunology;2021-07-29

4. Human Flexor Tendon Tissue Engineering: Decellularization of Human Flexor Tendons Reduces Immunogenicity In Vivo;Tissue Engineering Part A;2012-04

5. Failure of Effector Function of Human CD8+ T Cells in NOD/SCID/JAK3−/− Immunodeficient Mice Transplanted with Human CD34+ Hematopoietic Stem Cells;PLoS ONE;2010-10-01