HPV16 E6/E7 stabilize PGK1 protein by reducing its poly‐ubiquitination in cervical cancer
Author:
Affiliation:
1. Department of Obstetrics and Gynecology Cangzhou Central Hospital Cangzhou China
Publisher
Wiley
Subject
Cell Biology,General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/cbin.11744
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3. Adipose‐derived mesenchymal stem cells promote the malignant phenotype of cervical cancer;Castro‐Oropeza R.;Scientific Reports,2020
4. MicroRNA‐450b‐3p inhibits cell growth by targeting phosphoglycerate kinase 1 in hepatocellular carcinoma;Chen Z.;Journal of Cellular Biochemistry,2019
5. Functional polarization of tumour‐associated macrophages by tumour‐derived lactic acid;Colegio O. R.;Nature,2014
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2. Construction of a ferroptosis and hypoxia-related gene signature in cervical cancer to assess tumour immune microenvironment and predict prognosis;Journal of Obstetrics and Gynaecology;2024-02-29
3. Conduction and validation of a novel prognostic signature in cervical cancer based on the necroptosis characteristic genes via integrating of multiomics data;Computers in Biology and Medicine;2024-01
4. HPV E6/E7: insights into their regulatory role and mechanism in signaling pathways in HPV-associated tumor;Cancer Gene Therapy;2023-12-15
5. Cellular Retinoic Acid Binding Protein 2 (CRABP2), Up-regulated by HPV E6/E7, Leads to Aberrant Activation of the Integrin β1/FAK/ERK Signaling Pathway and Aggravates the Malignant Phenotypes of Cervical Cancer;Biochemical Genetics;2023-11-24
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