The p53 tumor suppressor participates in multiple cell cycle checkpoints
Author:
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcp.20689/fullpdf
Reference115 articles.
1. Interaction between replication protein A and p53 is disrupted after UV damage in a DNA repair-dependent manner
2. The p53-regulated Cyclin-dependent Kinase Inhibitor, p21 (cip1, waf1, sdi1), Is Not Required for Global Genomic and Transcription-coupled Nucleotide Excision Repair of UV-induced DNA Photoproducts
3. Distinct Initiation and Maintenance Mechanisms Cooperate to Induce G1 Cell Cycle Arrest in Response to DNA Damage
4. p53 controls both the G2/M and the G1 cell cycle checkpoints and mediates reversible growth arrest in human fibroblasts.
5. Maintenance of genomic integrity by p53: complementary roles for activated and non-activated p53
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