Rare Diseases That Impersonate One Another: X‐Linked Hypophosphatemia and Tumor‐Induced Osteomalacia, a Retrospective Analysis of Discriminating Features

Author:

DeCorte Joseph1ORCID,Randazzo Ericka1ORCID,Black Margo2ORCID,Hendrickson Chase3ORCID,Dahir Kathryn2ORCID

Affiliation:

1. Vanderbilt Medical Scientist Training Program Vanderbilt University Medical Center, Vanderbilt University School of Medicine Nashville TN USA

2. Program for Metabolic Bone Disorders at Vanderbilt, Division of Diabetes, Endocrinology, and Metabolism, Department of Medicine Vanderbilt University Medical Center Nashville TN USA

3. Division of Diabetes, Endocrinology, and Metabolism, Department of Medicine Vanderbilt University Medical Center Nashville TN USA

Funder

National Institute of General Medical Sciences

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Reference26 articles.

1. Evidence for FGF23 involvement in a bone‐kidney axis regulating bone mineralization and systemic phosphate and vitamin D homeostasis;Martin A;Adv Exp Med Biol,2012

2. Elevated fibroblast growth factor‐23 in hypophosphatemic linear nevus sebaceous syndrome;Hoffman WH;Am J Med Genet A,2005

3. Fibrous dysplasia, phosphate wasting and fibroblast growth factor 23;Imel EA;Pediatr Endocrinol Rev,2007

4. Tumor‐induced osteomalacia;Yin Z;Osteoporos Sarcop,2018

5. Phosphaturic mesenchymal tumors: a review and update;Folpe AL;Semin Diagn Pathol.,2019

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