Mechanisms of bone fragility in a mouse model of glucocorticoid-treated rheumatoid arthritis: Implications for insufficiency fracture risk-Reference-Cited by-同舟云学术

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Mechanisms of bone fragility in a mouse model of glucocorticoid-treated rheumatoid arthritis: Implications for insufficiency fracture risk

Published:2012-10-27 Issue:11 Volume:64 Page:3649-3659
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis & Rheumatism

Author:

Takahata Masahiko,Maher Jason R.,Juneja Subhash C.,Inzana Jason,Xing Lianping,Schwarz Edward M.,Berger Andrew J.,Awad Hani A.

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/art.34639/fullpdf

Reference51 articles.

1. High prevalence of asymptomatic vertebral fractures in post-menopausal women receiving chronic glucocorticoid therapy: a cross-sectional outpatient study;Angeli;Bone,2006

2. A meta-analysis of prior corticosteroid use and fracture risk;Kanis;J Bone Miner Res,2004

3. Characteristics of fracture and related factors in patients with rheumatoid arthritis;Nampei;Mod Rheumatol,2008

4. Inflammatory mediators as essential elements in bone remodeling;Goldring;Calcif Tissue Int,2003

5. Ubiquitin ligase Smurf1 mediates tumor necrosis factor-induced systemic bone loss by promoting proteasomal degradation of bone morphogenetic signaling proteins;Guo;J Biol Chem,2008

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