IL‐18R signaling is required for γδ T cell response and confers resistance to Trypanosoma cruzi infection-Reference-Cited by-同舟云学术

IL‐18R signaling is required for γδ T cell response and confers resistance to Trypanosoma cruzi infection

Published:2020-05-25 Issue:4 Volume:108 Page:1239-1251
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:J Leukoc Biol

Author:

da Mota Julia Barbalho¹,Echevarria‐Lima Juliana²^ORCID,Kyle‐Cezar Fernanda³,Melo Matheus¹,Bellio Maria⁴^ORCID,Scharfstein Julio¹^ORCID,Oliveira Ana Carolina¹^ORCID

Affiliation:

1. Laboratório de Imunologia Celular e Molecular, Instituto de Biofísica Carlos Chagas Filho Universidade Federal do Rio de Janeiro Rio de Janeiro Brazil

2. Laboratório de Imunologia Básica e Aplicada, Instituto de Microbiologia Paulo de Góes Universidade Federal do Rio de Janeiro Rio de Janeiro Brazil

3. Peter Gorer Department of Immunobiology, School of Immunology & Microbial Sciences King's College London London UK

4. Laboratório de Imunobiologia, Departamento de Imunologia, Instituto de Microbiologia Paulo de Góes Universidade Federal do Rio de Janeiro Rio de Janeiro Brazil

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/JLB.4MA0420-568R

Reference47 articles.

1. Acute Chagas Disease: New Global Challenges for an Old Neglected Disease

2. Defective Nitric Oxide Effector Functions Lead to Extreme Susceptibility of Trypanosoma cruzi -Infected Mice Deficient in Gamma Interferon Receptor or Inducible Nitric Oxide Synthase

3. Trypanosoma cruzi induces strong IL-12 and IL-18 gene expression in vivo: correlation with interferon-gamma (IFN-γ) production

4. IL-12 synergizes with IL-18 or IL-1β for IFN-γ production from human T cells

5. Early IFN-γ production is related to the presence of interleukin (IL)-18 and the absence of IL-13 in experimental Trypanosoma cruzi infections

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