Anti‐streptococcal antibody and T‐cell interactions with vascular endothelial cells initiate the development of rheumatic carditis-Reference-Cited by-同舟云学术

Anti‐streptococcal antibody and T‐cell interactions with vascular endothelial cells initiate the development of rheumatic carditis

Published:2019-10-16 Issue:2 Volume:107 Page:263-271
ISSN:0741-5400
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:J Leukoc Biol

Author:

Sikder Suchandan¹²³,Rush Catherine M.¹,Govan Brenda L.¹,Alim Md A.¹²,Ketheesan Natkunam⁴^ORCID

Affiliation:

1. College of Public Health Medical and Veterinary Sciences Australian Institute of Tropical Health and Medicine James Cook University Townsville Australia

2. Current affiliation: Chattogram Veterinary and Animal Sciences University Khulshi Chattogram Bangladesh

3. Current affiliation: School of Science and Technology University of New England Armidale Australia

4. School of Science and Technology University of New England Armidale Australia

Funder

National Health and Medical Research Council

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/JLB.4MA0919-096RR

Reference37 articles.

1. Cytotoxic mAb from rheumatic carditis recognizes heart valves and laminin

2. Repeat exposure to group A streptococcal M protein exacerbates cardiac damage in a rat model of rheumatic heart disease

3. Group G Streptococcus Induces an Autoimmune Carditis Mediated by Interleukin 17A and Interferon γ in the Lewis Rat Model of Rheumatic Heart Disease

4. Priming the Immune System for Heart Disease: A Perspective on Group A Streptococci

5. Pathogenic Mechanisms in Rheumatic Carditis: Focus on Valvular Endothelium

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