High glucose-induced Galectin-1 in human podocytes implicates the involvement of Galectin-1 in diabetic nephropathy
Author:
Affiliation:
1. Department of Nephrology; Second Xiangya Hospital; Central South University; Changsha Hunan P. R. China
2. Department of Pathology; University of lowa; Iowa City Iowa USA
3. Department of Nephrology; Yiyang Central Hospital; Hunan P. R. China
Publisher
Wiley
Subject
Cell Biology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/cbin.10363/fullpdf
Reference21 articles.
1. Nephrin and Podocin functions are highly conserved between the zebrafish pronephros and mammalian metanephros;Fukuyo;Mol Med Rep,2014
2. Urinary podocalyxin is an early marker for podocyte injury in patients with diabetes: establishment of a highly sensitive ELISA to detect urinary podocalyxin;Hara;Diabetologia,2012
3. Galectins in kidney development;Hughes;Glycoconj J,2002
4. MTORC1 activation in podocytes is a critical step in the development of diabetic nephropathy in mice;Inoki;J Clin Invest,2011
5. A glimpse of various pathogenetic mechanisms of diabetic nephropathy;Kanwar;Annu Rev Pathol,2011
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