Infiltrating monocytes versus resident kupffer cells: Do alternatively activated macrophages need to be targeted alternatively?
Author:
Publisher
Wiley
Subject
Hepatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/hep.24714/fullpdf
Reference15 articles.
1. Monocytes and macrophages as cellular targets in liver fibrosis;Heymann;Inflamm Allergy Drug Targets,2009
2. Kupffer cell heterogeneity: functional properties of bone marrow derived and sessile hepatic macrophages;Klein;Blood,2007
3. CX3CL1-CX3CR1 interaction prevents carbon tetrachloride-induced liver inflammation and fibrosis in mice;Aoyama;HEPATOLOGY,2010
4. Hepatic recruitment of the inflammatory Gr1+ monocyte subset upon liver injury promotes hepatic fibrosis;Karlmark;HEPATOLOGY,2009
5. The fractalkine receptor CX3CR1 protects against liver fibrosis by controlling differentiation and survival of infiltrating hepatic monocytes;Karlmark;HEPATOLOGY,2010
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4. Gulf war illness-related chemicals increase CD11b/c+ monocyte infiltration into the liver and aggravate hepatic cholestasis in a rodent model;Scientific Reports;2018-09-03
5. Differential Effects of Kupffer Cell Inactivation on Inflammation and The Liver Sieve Following Caecal-Ligation and Puncture-Induced Sepsis in Mice;Shock;2017-04
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