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Endoderm-specific deletion ofTbx1reveals an FGF-independent role for Tbx1 in pharyngeal apparatus morphogenesis

  • Published:2014-06-12 Issue:9 Volume:243 Page:1143-1151
  • ISSN:1058-8388
  • Container-title:Developmental Dynamics
  • language:en
  • Short-container-title:Dev. Dyn.

Author:

Jackson Abigail1,Kasah Sahrunizam1,Mansour Suzanne L.2,Morrow Bernice3,Basson M. Albert1

Affiliation:

1. Department of Craniofacial Development and Stem Cell Biology; King's College; London United Kingdom

2. Department of Human Genetics; University of Utah; Salt Lake City Utah

3. Department of Genetics; Albert Einstein College of Medicine; Bronx New York

Funder

Medical Research Council

National Institutes of Health

Publisher

Wiley

Subject

Developmental Biology

Reference46 articles.

1. Fgf8 is required for pharyngeal arch and cardiovascular development in the mouse;Abu-Issa;Development,2002

2. Dissection of Tbx1 and Fgf interactions in mouse models of 22q11DS suggests functional redundancy;Aggarwal;Hum Mol Genet,2006

3. Inactivation of Tbx1 in the pharyngeal endoderm results in 22q11DS malformations;Arnold;Development,2006

4. Begbie J Brunet JF Rubenstein JL Graham A. 1999 Induction of the epibranchial placodes. Development 126:895-902

5. Cre- mediated excision of Fgf8 in the Tbx1 expression domain reveals a critical role for Fgf8 in cardiovascular development in the mouse;Brown;Dev Biol,2004

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