Mitochondrial dysfunction in skeletal muscle contributes to the development of acute insulin resistance in mice-Reference-Cited by-同舟云学术

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Mitochondrial dysfunction in skeletal muscle contributes to the development of acute insulin resistance in mice

Published:2021-10-03 Issue:6 Volume:12 Page:1925-1939
ISSN:2190-5991
Container-title:Journal of Cachexia, Sarcopenia and Muscle
language:en
Short-container-title:J cachexia sarcopenia muscle

Author:

Lee Hyunjung¹,Ha Tae Youl¹²,Jung Chang Hwa¹²,Nirmala Farida Sukma¹²,Park So‐Young³,Huh Yang Hoon⁴,Ahn Jiyun¹²^ORCID

Affiliation:

1. Research Group of Natural Material and Metabolism Korea Food Research Institute Wanju Republic of Korea

2. Department of Food Biotechnology University of Science and Technology Daejeon Republic of Korea

3. Department of Physiology, College of Medicine Yeungnam University Daegu Republic of Korea

4. Center for Electron Microscopy Research Korea Basic Science Institute Cheongju Republic of Korea

Funder

Korea Food Research Institute

Publisher

Wiley

Subject

Physiology (medical),Orthopedics and Sports Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcsm.12794

Reference64 articles.

1. The pathogenesis of insulin resistance: integrating signaling pathways and substrate flux

2. Molecular Mechanisms of Insulin Resistance in Humans and Their Potential Links With Mitochondrial Dysfunction

3. Impaired Mitochondrial Activity in the Insulin-Resistant Offspring of Patients with Type 2 Diabetes

4. Sleep Fragmentation in Mice Induces Nicotinamide Adenine Dinucleotide Phosphate Oxidase 2-Dependent Mobilization, Proliferation, and Differentiation of Adipocyte Progenitors in Visceral White Adipose Tissue

5. Cellular alterations in liver, skeletal muscle, and adipose tissue responsible for insulin resistance in obesity and type II diabetes

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