Opening of the CLC‐3 chloride channel induced by dihydroartemisinin contributed to early apoptotic events in human poorly differentiated nasopharyngeal carcinoma cells-Reference-Cited by-同舟云学术

Opening of the CLC‐3 chloride channel induced by dihydroartemisinin contributed to early apoptotic events in human poorly differentiated nasopharyngeal carcinoma cells

Published:2018-09 Issue:11 Volume:119 Page:9560-9572
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Zhou Congran¹,Tang Xinwei¹,Xu Jingkui²,Wang Jiajia¹,Yang Yaping³,Chen Yehui⁴,Chen Lixin¹,Wang Liwei²,Zhu Linyan¹^ORCID,Yang Haifeng⁵

Affiliation:

1. Department of Pharmacology School of Medicine, Jinan University Guangzhou China

2. Department of Physiology School of Medicine, Jinan University Guangzhou China

3. Analysis and Test Center Jinan University Guangzhou China

4. Department of Urology Guangzhou First People’s Hospital, Guangzhou Medical University Guangzhou China

5. Department of Pathology Guangdong Provincial Hospital of Chinese Medicine, The Second Affiliated Hospital of Guangzhou University of Chinese Medicine Guangzhou China

Funder

Natural Science Foundation of Guangdong Province

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.27274

Reference35 articles.

1. Anticancer activity of artemisinin and its derivatives;Slezakova S;Anticancer Res,2017

2. Interruption of the MEK/ERK signaling cascade promotes dihydroartemisinin-induced apoptosis in vitro and in vivo