Loss of <i>CDKN1B</i> induces an age‐related clonal hematopoietic disorder via Notch2 activity dysregulation-Reference-Cited by-同舟云学术

Loss of CDKN1B induces an age‐related clonal hematopoietic disorder via Notch2 activity dysregulation

Published:2023-06-19 Issue:7 Volume:43 Page:844-849
ISSN:2523-3548
Container-title:Cancer Communications
language:en
Short-container-title:Cancer Communications

Author:

Segatto Ilenia¹^ORCID,Rampioni Vinciguerra Gian Luca¹²^ORCID,Pellarin Ilenia¹^ORCID,Dall'Acqua Alessandra¹^ORCID,Berton Stefania¹^ORCID,Citron Francesca¹^ORCID,D'Andrea Sara¹,Mungo Giorgia¹,Viotto Davide¹,Musco Lorena¹^ORCID,Di Napoli Arianna²,Aloe Spiriti Maria Antonietta²,Canzonieri Vincenzo³,Gattei Valter⁴,Vecchione Andrea²,Belletti Barbara¹^ORCID,Baldassarre Gustavo¹^ORCID

Affiliation:

1. Division of Molecular Oncology Centro di Riferimento Oncologico di Aviano (CRO) Istituto di Ricovero e Cura a Carattere Scientifico (IRCCS) National Cancer Institute Aviano (PN) Italy

2. Department of Clinical and Molecular Medicine University of Rome “La Sapienza” Santo Andrea Hospital Rome Italy

3. Division of Pathology Centro di Riferimento Oncologico di Aviano (CRO) Istituto di Ricovero e Cura a Carattere Scientifico (IRCCS) National Cancer Institute Aviano (PN) Italy

4. Division of Clinical and Experimental Onco‐Hematology Centro di Riferimento Oncologico di Aviano (CRO) Istituto di Ricovero e Cura a Carattere Scientifico (IRCCS) National Cancer Institute Aviano (PN) Italy

Publisher

Wiley

Subject

Cancer Research,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cac2.12444

Reference11 articles.

1. TEL and KIP1 define the smallest region of deletions on 12p13 in hematopoietic malignancies

2. Genetic predisposition to MDS: clinical features and clonal evolution

3. Myelodysplastic syndrome progression to acute myeloid leukemia at the stem cell level

4. Turning the tide in myelodysplastic/myeloproliferative neoplasms

5. A Syndrome of Multiorgan Hyperplasia with Features of Gigantism, Tumorigenesis, and Female Sterility in p27Kip1-Deficient Mice

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