Elucidation of pathological mechanism caused by human disease mutation in CaMKIIβ-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Elucidation of pathological mechanism caused by human disease mutation in CaMKIIβ

Published:2022-01-18 Issue:3 Volume:100 Page:880-896
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J of Neuroscience Research

Author:

Mutoh Hiroki¹^ORCID,Aoto Kazushi¹,Miyazaki Takehiro¹²,Fukuda Atsuo³,Saitsu Hirotomo¹^ORCID

Affiliation:

1. Department of Biochemistry Hamamatsu University School of Medicine Hamamatsu Japan

2. Department of Molecular Genetics, Graduate School of Medicine Kyoto University Kyoto Japan

3. Department of Neurophysiology Hamamatsu University School of Medicine Hamamatsu Japan

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.25013

Reference43 articles.

1. De novo variants in CAMK 2A and CAMK 2B cause neurodevelopmental disorders

2. Generation and Behavior Characterization of CaMKIIβ Knockout Mice

3. Developmental expression of the CaM kinase II isoforms: ubiquitous γ- and δ-CaM kinase II are the early isoforms and most abundant in the developing nervous system

4. CaM Kinase: Still Inspiring at 40

5. CaMKII Plays a Nonenzymatic Role in Hippocampal Synaptic Plasticity and Learning by Targeting CaMKII to Synapses

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cnpy3 mice reveal neuronal expression of Cnpy3 in the brain;Journal of Neuroscience Methods;2023-01

2. Generation of Flag/DYKDDDDK Epitope Tag Knock-In Mice Using i-GONAD Enables Detection of Endogenous CaMKIIα and β Proteins;International Journal of Molecular Sciences;2022-10-07

3. Förster resonance energy transfer-based kinase mutation phenotyping reveals an aberrant facilitation of Ca2+/calmodulin-dependent CaMKIIα activity in de novo mutations related to intellectual disability;Frontiers in Molecular Neuroscience;2022-09-01

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3