Reversal of increased lymphocyte PC-1 activity in patients with Type 2 diabetes treated with metformin
Author:
Publisher
Wiley
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Reference25 articles.
1. The Triumvirate: β-Cell, Muscle, Liver: A Collusion Responsible for NIDDM
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3. Mechanisms of the Kinetic Defect in Insulin Action in Obesity and NIDDM
4. PC-1 content in skeletal muscle of non-obese, non-diabetic subjects: relationship to insulin receptor tyrosine kinase and whole body insulin sensitivity
5. Increased adipose tissue PC-1 protein content, but not tumour necrosis factor-a gene expression, is associated with a reduction of both whole body insulin sensitivity and insulin receptor tyrosine-kinase activity
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