High levels of iron supplementation prevents neural tube defects in theFpn1ffemouse model

Author:

Stokes Bethany A.12,Sabatino Julia A.2,Zohn Irene E.2

Affiliation:

1. Department of Biology; The George Washington University; Washington DC

2. Center for Neuroscience Research, Children's Research Institute, Children's National Medical Center; Washington DC

Funder

Board of Visitors to the Children's National Medical Center

Eunice Kennedy Shriver National Institute of Child Health & Human Development

National Center for Research Resources

CRI and NIH

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology,Pediatrics, Perinatology and Child Health

Reference77 articles.

1. Visceral endoderm-restricted translation of Otx1 mediates recovery of Otx2 requirements for specification of anterior neural plate and normal gastrulation;Acampor;Development,1998

2. Folic acid deficiency in hemochromatosis: probably due to a defective storage of folic acid in the liver;Arakawa;Tohoku J Exp Med,1965

3. Neural tube defects and folate: case far from closed;Blom;Nat Rev,2006

4. Iron requirements in pregnancy and strategies to meet them;Bothwell;Am J Clin Nutr,2000

5. Maternal zinc status: a determination of central nervous system malformation;Buamah;Br J Obstet Gynaecol,1984

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