Angiogenesis is repressed by ethanol exposure during chick embryonic development

Author:

Wang Guang12,Zhong Shan1,Zhang Shi-yao1,Ma Zheng-lai1,Chen Jian-long1,Lu Wen-hui1,Cheng Xin1,Chuai Manli3,Lee Kenneth Ka Ho4,Lu Da-xiang2,Yang Xuesong1

Affiliation:

1. Division of Histology and Embryology, Key Laboratory for Regenerative Medicine of the Ministry of Education, Medical College; Jinan University; Guangzhou People's Republic of China

2. Department of Pathophysiology, Institute of Brain Research, Medical College; Jinan University; Guangzhou People's Republic of China

3. Division of Cell and Developmental Biology; University of Dundee; Dundee UK

4. Key Laboratory for Regenerative Medicine of the Ministry of Education, School of Biomedical Sciences; Chinese University of Hong Kong; Shatin Hong Kong

Funder

NSFC

China Postdoctoral Science Foundation

Science and Technology Program of Guangzhou

Fundamental Research Funds for the Central Universities

Funds for Young Creative Talents of Higher Education in Guangdong Province; Chinese Medicine Science and Technology Program of Guangdong Province

Publisher

Wiley

Subject

Toxicology

Reference48 articles.

1. Prenatal effects of alcohol;Abel;Drug Alcohol Depend.,1984

2. An update on incidence of FAS: FAS is not an equal opportunity birth defect;Abel;Neurotoxicol. Teratol.,1995

3. Comparison of in vitro and in utero ethanol exposure on indices of oxidative stress;Akella;In Vitro Mol. Toxicol.,2000

4. Acute and chronic ethanol increases reactive oxygen species generation and decreases viability in fresh, isolated rat hepatocytes;Bailey;Hepatology,1998

5. Microglia play a role in ethanol-induced oxidative stress and apoptosis in developing hypothalamic neurons;Boyadjieva;Alcohol. Clin. Exp. Res.,2013

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