Bisphenol S favors hepatic steatosis development via an upregulation of liver MCT1 expression and an impairment of the mitochondrial respiratory system-Reference-Cited by-同舟云学术

Bisphenol S favors hepatic steatosis development via an upregulation of liver MCT1 expression and an impairment of the mitochondrial respiratory system

Published:2022-05-13 Issue:7 Volume:237 Page:3057-3068
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:Journal Cellular Physiology

Author:

Mornagui Bessem¹^ORCID,Rezg Raja²^ORCID,Repond Cendrine³^ORCID,Pellerin Luc³⁴^ORCID

Affiliation:

1. Faculty of Sciences of Gabes University of Gabes Gabes Tunisia

2. Higher Institute of Biotechnology of Monastir University of Monastir Monastir Tunisia

3. Department of Physiology University of Lausanne Lausanne Switzerland

4. Inserm U1313, Faculté de Médecine et de Pharmacie Université et CHU de Poitiers Poitiers Cedex France

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.30771

Reference74 articles.

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2. Metabolic disruption in male mice due to fetal exposure to low but not high doses of bisphenol A (BPA): Evidence for effects on body weight, food intake, adipocytes, leptin, adiponectin, insulin and glucose regulation

3. Superoxide dismutase: Improved assays and an assay applicable to acrylamide gels

4. Nonalcoholic Fatty Liver Disease

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