Proanthocyanidins attenuate the high glucose‐induced damage of retinal pigment epithelial cells by attenuating oxidative stress and inhibiting activation of the NLRP3 inflammasome-Reference-Cited by-同舟云学术

Proanthocyanidins attenuate the high glucose‐induced damage of retinal pigment epithelial cells by attenuating oxidative stress and inhibiting activation of the NLRP3 inflammasome

Published:2021-08-02 Issue:9 Volume:35 Page:
ISSN:1095-6670
Container-title:Journal of Biochemical and Molecular Toxicology
language:en
Short-container-title:J Biochem Mol Toxicol

Author:

Li Hongsong¹^ORCID,Li Rong²^ORCID,Wang Lijun¹,Liao Dingying¹,Zhang Wenyi¹,Wang Jianming¹^ORCID

Affiliation:

1. Department of Ophthalmology The Second Affiliated Hospital of Xi'an Jiaotong University Xi'an China

2. Department of Ophthalmology The First Affiliated Hospital of Xi'an Medical University Xi'an China

Funder

Key Research and Development Projects of Shaanxi Province

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Toxicology,Molecular Biology,Molecular Medicine,Biochemistry,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbt.22845

Reference32 articles.

1. Diabetic retinopathy

2. Osteomeles schwerinae Extract and Its Major Compounds Inhibit Methylglyoxal-Induced Apoptosis in Human Retinal Pigment Epithelial Cells

3. Advanced glycation end-product (AGE) induces apoptosis in human retinal ARPE-19 cells via promoting mitochondrial dysfunction and activating the Fas-FasL signaling

4. The Oxidative Stress and Mitochondrial Dysfunction during the Pathogenesis of Diabetic Retinopathy

5. Oxidative stress and epigenetic modifications in the pathogenesis of diabetic retinopathy

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