Reduced N6‐Methyladenosine Mediated by METTL3 Acetylation Promotes MTF1 Expression and Hepatocellular Carcinoma Cell Growth-Reference-Cited by-同舟云学术

Reduced N6‐Methyladenosine Mediated by METTL3 Acetylation Promotes MTF1 Expression and Hepatocellular Carcinoma Cell Growth

Published:2022-10-17 Issue:11 Volume:19 Page:
ISSN:1612-1872
Container-title:Chemistry & Biodiversity
language:en
Short-container-title:Chemistry & Biodiversity

Author:

Yang Ying¹^ORCID,Qian Cai Qian¹,Sheng Fu Li¹,Wei Dong Yi¹,Fan Fei²,Zhong Wu Xing¹^ORCID

Affiliation:

1. Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences Fudan University 138 Yi Xue Yuan Road Shanghai 200032 P. R. China

2. Department of the 3rd Ward of Special Treatment Shanghai Eastern Hepatobiliary Surgery Hospital Shanghai 200438 P. R. China

Funder

National Natural Science Foundation of China

Shanghai Municipal Health Commission

Publisher

Wiley

Subject

Molecular Biology,Molecular Medicine,General Chemistry,Biochemistry,General Medicine,Bioengineering

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cbdv.202200333

Reference32 articles.

1. Gene expression regulation mediated through reversible m6A RNA methylation

2. RNA N6-methyladenosine methyltransferase-like 3 promotes liver cancer progression through YTHDF2-dependent posttranscriptional silencing of SOCS2

3. Single-nucleotide-resolution mapping of m6A and m6Am throughout the transcriptome

4. Topology of the human and mouse m6A RNA methylomes revealed by m6A-seq

5. The m 6 A Methyltransferase METTL3 Promotes Translation in Human Cancer Cells

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