Genomic instability: Crossing pathways at the origin of structural and numerical chromosome changes

Author:

Russo Antonella1,Pacchierotti Francesca2,Cimini Daniela3,Ganem Neil J.4,Genescà Anna5,Natarajan Adayapalam T.6,Pavanello Sofia7,Valle Giorgio1,Degrassi Francesca8

Affiliation:

1. Department of Biology; University of Padova; Padova Italy

2. Laboratory of Toxicology; Unit of Radiation Biology and Human Health; ENEA CR Casaccia Rome Italy

3. Department of Biological Sciences and Virginia Bioinformatics Institute; Virginia Tech Blacksburg Virginia

4. Department of Pharmacology, Division of Hematology and Oncology; Boston University School of Medicine; Boston Massachusetts

5. Department of Cell Biology, Physiology and Immunology; Universitat Autònoma de Barcelona; Bellaterra Spain

6. Department of Ecological and Biological Sciences; University of Tuscia; Viterbo Italy

7. Department of Cardiac, Thoracic and Vascular Sciences, Unit of Occupational Medicine; University of Padova; Italy

8. IBPM; Institute of Molecular Biology and Pathology; CNR Rome Italy

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Genetics(clinical),Epidemiology

Reference160 articles.

1. Death without caspases, caspases without death;Abraham;Trends Cell Biol,2004

2. Implementing toxicity testing in the 21st century (TT21C): Making safety decisions using toxicity pathways, and progress in a prototype risk assessment;Adeleye;Toxicology

3. The source and significance of DNA damage in human spermatozoa; a commentary on diagnostic strategies and straw man fallacies;Aitken;Mol Hum Reprod,2013

4. Tetraploid state induces p53-dependent arrest of nontransformed mammalian cells in G1;Andreassen;Mol Biol Cell,2001

5. Mismatch repair in correction of replication errors and processing of DNA damage;Aquilina;J Cell Physiol,2001

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