Tyrosine kinase inhibitor (TKI)-induced cardiotoxicity: approaches to narrow the gaps between preclinical safety evaluation and clinical outcome-Reference-Cited by-同舟云学术

Tyrosine kinase inhibitor (TKI)-induced cardiotoxicity: approaches to narrow the gaps between preclinical safety evaluation and clinical outcome

Published:2012-09-10 Issue:12 Volume:32 Page:945-951
ISSN:0260-437X
Container-title:Journal of Applied Toxicology
language:en
Short-container-title:J. Appl. Toxicol.

Author:

Yang Baichun¹,Papoian Thomas¹

Affiliation:

1. Center for Drug Evaluation and Research; Food and Drug Administration; 10903 New Hampshire Avenue; Silver Spring; MD; 20993; USA

Publisher

Wiley

Subject

Toxicology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jat.2813/fullpdf

Reference51 articles.

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2. Sunitinib, a receptor tyrosine kinase inhibitor, increases blood pressure in rats without associated changes in cardiac structure and function;Blasi;Cardiovasc. Ther.,2011

3. Prediction of drug-induced cardiotoxicity using human embryonic stem cell-derived cardiomyocytes;Braam;Stem Cell Res.,2010

4. Mechanisms of cardiac dysfunction associated with tyrosine kinase inhibitor cancer therapeutics;Chen;Circulation,2008

5. Molecular mechanisms of cardiovascular toxicity of targeted cancer therapeutics;Cheng;Circ. Res.,2010

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