Conjugated Linoleic Acid and Alpha Linolenic Acid Improve Cholesterol Homeostasis in Obesity by Modulating Distinct Hepatic Protein Pathways-Reference-Cited by-同舟云学术

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Conjugated Linoleic Acid and Alpha Linolenic Acid Improve Cholesterol Homeostasis in Obesity by Modulating Distinct Hepatic Protein Pathways

Published:2020-04 Issue:7 Volume:64 Page:1900599
ISSN:1613-4125
Container-title:Molecular Nutrition & Food Research
language:en
Short-container-title:Mol. Nutr. Food Res.

Author:

O'Reilly Marcella E.¹²^ORCID,Lenighan Yvonne M.¹²,Dillon Eugene³²,Kajani Sarina⁴²⁵,Curley Sean⁴²⁵,Bruen Robyn⁴²,Byrne Rachel⁴²⁵,Heslin Aoibhin Moore¹²,Moloney Aidan P.⁶,Roche Helen M.¹⁷²,McGillicuddy Fiona C.⁴²⁵^ORCID

Affiliation:

1. Nutrigenomics Research Group, School of Public Health Physiotherapy and Sports ScienceUniversity College Dublin Dublin 4 Ireland

2. UCD Conway InstituteUniversity College Dublin Dublin 4 Ireland

3. Mass Spectrometry ResourceUniversity College Dublin Dublin 4 Ireland

4. Diabetes Complications Research CentreUniversity College Dublin Dublin 4 Ireland

5. UCD School of MedicineUniversity College Dublin Dublin 4 Ireland

6. TeagascAnimal & Grassland Research and Innovation Centre Meath Ireland

7. UCD Institute of Food and HealthUniversity College Dublin Dublin 4 Ireland

Funder

Science Foundation Ireland

Publisher

Wiley

Subject

Food Science,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/mnfr.201900599

Reference42 articles.

1. Effect of Insulin Resistance, Dyslipidemia, and Intra-abdominal Adiposity on the Development of Cardiovascular Disease and Diabetes Mellitus

2. Regulation of reverse cholesterol transport and clinical implications

3. High-Density Lipoprotein Proteomic Composition, and not Efflux Capacity, Reflects Differential Modulation of Reverse Cholesterol Transport by Saturated and Monounsaturated Fat Diets

4. Cooperative transcriptional activation of ATP-binding cassette sterol transporters ABCG5 and ABCG8 genes by nuclear receptors including Liver-X-Receptor

5. Impaired expression and function of the bile salt export pump due to three novel ABCB11 mutations in intrahepatic cholestasis

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