The Role of Dietary Advanced Glycation End Products in Metabolic Dysfunction-Reference-Cited by-同舟云学术

The Role of Dietary Advanced Glycation End Products in Metabolic Dysfunction

Published:2020-04-20 Issue:1 Volume:65 Page:1900934
ISSN:1613-4125
Container-title:Molecular Nutrition & Food Research
language:en
Short-container-title:Mol. Nutr. Food Res.

Author:

Sergi Domenico¹²,Boulestin Hakim³,Campbell Fiona M.³,Williams Lynda M.³

Affiliation:

1. Nutrition and Health Substantiation Group Nutrition and Health Program Health and Biosecurity Commonwealth Scientific and Industrial Research Organisation (CSIRO) Adelaide SA 5000 Australia

2. Adelaide Medical School The University of Adelaide Adelaide SA 5000 Australia

3. Rowett Institute University of Aberdeen Aberdeen AB25 2ZD UK

Funder

Rural and Environment Science and Analytical Services Division

Commonwealth Scientific and Industrial Research Organisation

Publisher

Wiley

Subject

Food Science,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/mnfr.201900934

Reference150 articles.

1. Dietary sugars, not lipids, drive hypothalamic inflammation

2. Immunohistochemical colocalization of glycoxidation products and lipid peroxidation products in diabetic renal glomerular lesions. Implication for glycoxidative stress in the pathogenesis of diabetic nephropathy.

3. Increased accumulation of the glycoxidation product N(epsilon)-(carboxymethyl)lysine in human tissues in diabetes and aging.

4. ChREBP regulates fructose-induced glucose production independently of insulin signaling

5. Formation of Fructose-Mediated Advanced Glycation End Products and Their Roles in Metabolic and Inflammatory Diseases

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