In vivoadministration of the C16:0 fatty acid isoform of sulfatide increases pancreatic sulfatide and enhances glucose-stimulated insulin secretion in Zucker fatty (fa/fa) rats-Reference-Cited by-同舟云学术

In vivoadministration of the C16:0 fatty acid isoform of sulfatide increases pancreatic sulfatide and enhances glucose-stimulated insulin secretion in Zucker fatty (fa/fa) rats

Published:2004-12-03 Issue:2 Volume:21 Page:158-166
ISSN:1520-7552
Container-title:Diabetes/Metabolism Research and Reviews
language:en
Short-container-title:Diabetes Metab. Res. Rev.

Author:

Blomqvist Maria,Carrier Martin,Andrews Tara,Pettersson Knut,Månsson Jan-Eric,Rynmark Britt-Marie,Fredman Pam,Buschard Karsten

Publisher

Wiley

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine

Reference40 articles.

1. Sulphatide antigen in islets of Langerhans and in diabetic glomeruli, and anti-sulphatide antibodies in type 1 diabetes mellitus;Buschard;APMIS,1993

2. Sulphatide in islets of Langerhans and in organs affected in diabetic late complications: a study in human and animal tissue;Buschard;Diabetologia,1994

3. The glycosphingolipid sulfatide in the islets of Langerhans in rat pancreas is processed through recycling; possible involvement in insulin trafficking;Fredman;Glycobiology,2000

4. Sulfatide promotes the folding of proinsulin, preserves insulin crystals, and mediates its monomerization;Osterbye;Glycobiology,2001

5. Sulfatide controls insulin secretion by modulation of ATP-sensitive K+-channel activity and Ca2+-dependent exocytosis in rat pancreatic beta-cells;Buschard;Diabetes,2002

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