Cellular Hypoxia Promotes Heterotopic Ossification by Amplifying BMP Signaling-Reference-Cited by-同舟云学术

Cellular Hypoxia Promotes Heterotopic Ossification by Amplifying BMP Signaling

Published:2016-04-20 Issue:9 Volume:31 Page:1652-1665
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Wang Haitao¹²,Lindborg Carter¹²,Lounev Vitali¹²,Kim Jung‐Hoon³,McCarrick‐Walmsley Ruth¹²,Xu Meiqi¹²,Mangiavini Laura⁴,Groppe Jay C⁵,Shore Eileen M¹²⁶,Schipani Ernestina⁴,Kaplan Frederick S¹²³,Pignolo Robert J¹²³

Affiliation:

1. Department of Orthopaedic Surgery, Perelman School of MedicineThe University of PennsylvaniaPhiladelphiaPAUSA

2. The Center for Research in FOP and Related Disorders, Perelman School of MedicineThe University of PennsylvaniaPhiladelphiaPAUSA

3. Department of Medicine, Perelman School of MedicineThe University of PennsylvaniaPhiladelphiaPAUSA

4. Departments of Medicine and Orthopaedic SurgeryUniversity of MichiganAnn ArborMIUSA

5. Department of Biomedical SciencesTexas A&M University Baylor College of DentistryDallasTXUSA

6. Department of Genetics, Perelman School of MedicineThe University of PennsylvaniaPhiladelphiaPAUSA

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.2848

Reference79 articles.

1. Skeletal metamorphosis in fibrodysplasia ossificans progressiva (FOP)

2. Nonhereditary Heterotopic Ossification: Implications for Injury, Arthropathy, and Aging

3. A recurrent mutation in the BMP type I receptor ACVR1 causes inherited and sporadic fibrodysplasia ossificans progressiva

4. Classic and atypical fibrodysplasia ossificans progressiva (FOP) phenotypes are caused by mutations in the bone morphogenetic protein (BMP) type I receptor ACVR1

5. Structure of the Bone Morphogenetic Protein Receptor ALK2 and Implications for Fibrodysplasia Ossificans Progressiva

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